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Dernières nouvelles
Inhibition of tyrosine phosphorylation by wortmannin in human neutrophils. Dissociation from its inhibitory effects on phospholipase D
Article de revueLab Invest, 69 (1), 1993.
Granulocyte-macrophage colony-stimulating factor primes phospholipase D activity in human neutrophils in vitro: role of calcium, G-proteins and tyrosine kinases
Article de revueCell Signal, 4 (5), 1992.
Granulocyte-macrophage colony-stimulating factor increases the synthesis of leukotriene B4 by human neutrophils in response to platelet-activating factor. Enhancement of both arachidonic acid availability and 5-lipoxygenase activation
Article de revueJ Immunol, 146 (4), 1991.
Involvement of a phospholipase D in the mechanism of action of granulocyte-macrophage colony-stimulating factor (GM-CSF): priming of human neutrophils in vitro with GM-CSF is associated with accumulation of phosphatidic acid and diradylglycerol
Article de revueJ Exp Med, 172 (3), 1990.
Automated on-line extraction and profiling of lipoxygenase products of arachidonic acid by high-performance liquid chromatography
Article de revueMethods Enzymol, 187 , 1990.
Altered formation of leukotriene B4 in vitro by synovial fluid neutrophils in rheumatoid arthritis
Article de revueJ Rheumatol, 16 (3), 1989.
Granulocyte-macrophage colony-stimulating factor (GM-CSF) and opsonization synergistically enhance leukotriene B4 (LTB4) synthesis induced by phagocytosis in human neutrophils
Article de revueAgents Actions, 27 (3-4), 1989.
Studies on the effects of disease-modifying antirheumatic drugs on lipoxygenase product synthesis in human neutrophils in vitro
Article de revueInt J Immunopharmacol, 10 (6), 1988.
Assessment of leukotriene B4 synthesis in human lymphocytes by using high performance liquid chromatography and radioimmunoassay methods
Article de revueJ Immunol, 139 (4), 1987.
Leukotriene synthesis by synovial fluid and blood polymorphonuclear leukocytes (PMNL) and monocytomacrophages of rheumatoid arthritis (RA)
Article de revue10 , 1986.
Projets actifs
- Metabolic mechanisms of pro-resolutive neutrophils in chronic inflammatory diseases, du 2024-04-01 au 2029-03-31